Repairing a 'bad' reputation?

New research at the Salk Institute for Biological Studies casts the role of a neuronal growth factor receptor—long suspected to facilitate the toxic effects of beta amyloid in Alzheimer's disease— in a new light, suggesting the molecule actually protects the neuron in the periphery from beta amyloid-induced damage.

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<p><a href="http://www.physorg.com/news159466987.html">Repairing a 'bad' reputation?</a></p> <p>New research at the Salk Institute for Biological Studies casts the role of a neuronal growth factor receptor—long suspected to facilitate the toxic effects of beta amyloid in Alzheimer's disease— in a new light, suggesting the molecule actually protects the neuron in the periphery from beta amyloid-induced damage.</p>