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     <title>Study supports DNA repair-blocker research in cancer therapy</title>
   	 <description>Scientists at Dana-Farber Cancer Institute have uncovered the mechanism behind a promising new approach to cancer treatment: damaging cancer cells' DNA with potent drugs while simultaneously preventing the cells from repairing themselves.</description>
     <link>http://www.physorg.com/news169725288.html</link>
	 <category>Medicine &amp; Health</category>
	 <pubDate>Mon, 17 Aug 2009 10:55:26 EST</pubDate>
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     <title>Firm providing US airport 'fast lanes' shuts down</title>
   	 <description>Clear, a company which pre-screened travellers and issued high-tech cards allowing them to whisk through security lines at US airports, is going out of business.</description>
     <link>http://www.physorg.com/news164981614.html</link>
	 <category>Technology</category>
	 <pubDate>Tue, 23 Jun 2009 13:13:59 EST</pubDate>
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     <title>Researchers illuminate mechanisms that regulate DNA damage control and replication</title>
   	 <description>Scientists at Burnham Institute for Medical Research (Burnham) have demonstrated important new roles for the protein kinase complex Cdc7/Dbf4 or Cdc7/Drf1 (Ddk) in monitoring damage control during DNA replication and reinitiating replication following DNA repair. Since Ddk is often deregulated in human cancers, this new understanding of its role in DNA damage control could help shape new cancer therapies. The research was published in the December 24 issue of Molecular Cell.</description>
     <link>http://www.physorg.com/news150397201.html</link>
	 <category>Biology</category>
	 <pubDate>Mon, 05 Jan 2009 17:00:01 EST</pubDate>
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     <title>Stem-cell sentry sounds the alarm to maintain balance between cancer and aging</title>
   	 <description>(PhysOrg.com) -- Like a sentry guarding the castle walls, a molecular messenger inside adult stem cells sounds the alarm when it senses hazards that could allow the invasion of an insidious enemy: Cancer. The alarm bell halts the process of cell division in its tracks, preventing an error that could lead to runaway cell division and eventually, tumor formation.</description>
     <link>http://www.physorg.com/news143295152.html</link>
	 <category>Medicine &amp; Health</category>
	 <pubDate>Wed, 15 Oct 2008 13:12:32 EST</pubDate>
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     <title>Scripps research team unravels new cellular repair mechanism</title>
   	 <description>A Scripps Research team has unraveled a new biochemical pathway that triggers a critical repair response to correct errors in the DNA replication process that could otherwise lead to harmful or fatal mutations in cells. Though the work focused on yeast cells, the team expects to find an analogous system in human cells that could be exploited as a target for potential therapies for cancers, which are often caused by such repair mechanisms going off course. The research was published today in an advanced, online issue of the Proceedings of the National Academy of Sciences.</description>
     <link>http://www.physorg.com/news137261363.html</link>
	 <category>Biology</category>
	 <pubDate>Wed, 06 Aug 2008 17:09:23 EST</pubDate>
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     <title>Death, division or cancer? Newly discovered checkpoint process holds the line in cell division</title>
   	 <description>Each day, a staggering number of cells perform a feat that still amazes researchers with its complexity: they divide to produce perfect replicas of each other. The process is called mitosis, and an inability to control it is one of the hallmarks of cancer.</description>
     <link>http://www.physorg.com/news134147252.html</link>
	 <category>Medicine &amp; Health</category>
	 <pubDate>Tue, 01 Jul 2008 16:07:32 EST</pubDate>
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