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<title>PHYSorg.com: PHYSorg news tagged with: glutamate receptor</title>
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 <item>
     <title>Researchers solve structure of NMDA receptor unit that could be drug target for neurological diseases</title>
   	 <description>A team of scientists at Cold Spring Harbor Laboratory reports on Thursday their success in solving the molecular structure of a key portion of a cellular receptor implicated in Alzheimer's, Parkinson's, and other serious illnesses.</description>
     <link>http://www.physorg.com/news177250907.html</link>
	 <category>Chemistry</category>
	 <pubDate>Thu, 12 Nov 2009 12:22:55 EST</pubDate>
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     <title>Understanding the brain's natural foil for over-excited neurons</title>
   	 <description>Glutamate is to the brain like coffee is to our bodies. A cup of Joe in the morning can wake us, but overloading on caffeine causes the stimulant to work against us.</description>
     <link>http://www.physorg.com/news175191974.html</link>
	 <category>Medicine &amp; Health</category>
	 <pubDate>Mon, 19 Oct 2009 19:20:01 EST</pubDate>
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     <title>Hitting cell hot spot could help thwart Parkinson's disease</title>
   	 <description>The latest work to 'turn off the taps' in the brain and stop a chemical being released in excess amounts - which can lead to Parkinson's Disease - will be presented at The British Pharmacological Society's Summer Meeting in Edinburgh today (Wednesday, 8 July 2009).</description>
     <link>http://www.physorg.com/news166251940.html</link>
	 <category>Medicine &amp; Health</category>
	 <pubDate>Wed, 08 Jul 2009 06:20:02 EST</pubDate>
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     <title>Flies avoid a plant's poison using a newly identified taste mechanism</title>
   	 <description>Many plants protect themselves from hungry animals by producing toxic chemicals. In turn, animals rely on detecting the presence of these harmful chemicals to avoid consuming dangerous plant material. A paper, published in this week's issue of PLoS Biology, investigates the response of an insect to a common plant weapon - the toxin L-canavanine.</description>
     <link>http://www.physorg.com/news165566601.html</link>
	 <category>Biology</category>
	 <pubDate>Tue, 30 Jun 2009 08:20:01 EST</pubDate>
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     <title>Discovery could help scientists stop the "death cascade" of neurons after a stroke</title>
   	 <description>(PhysOrg.com) -- Distressed swimmers often panic, sapping the strength they need to keep their heads above water until help arrives. When desperate for oxygen, neurons behave in a similar way. They freak out, stupidly discharging energy until they drown in a sea of their own extruded salts. Every year, millions of victims of stroke or brain trauma suffer permanent brain damage because of this mad rush to oblivion that begins once a part of the brain is deprived of blood.</description>
     <link>http://www.physorg.com/news151342666.html</link>
	 <category>Biology</category>
	 <pubDate>Fri, 16 Jan 2009 15:37:46 EST</pubDate>
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     <title>Glutamate:  Too much of a good thing in schizophrenia?</title>
   	 <description>Is schizophrenia a disorder of glutamate hyperactivity or hypoactivity?  While the predominant hypothesis for many years was that schizophrenia was a glutamate deficit disorder, there is growing evidence of glutamate hyperactivity as well.  The study by Karlsson et al., appearing in the November 1st issue of Biological Psychiatry, reinforces this point with new data about the impact of deleting the gene for the glutamate transporter EAAT1.  EAAT1, implicated in schizophrenia, plays a critical role in inactivating glutamate by removing it from the synaptic and extracellular spaces.  </description>
     <link>http://www.physorg.com/news144326615.html</link>
	 <category>Medicine &amp; Health</category>
	 <pubDate>Mon, 27 Oct 2008 11:43:35 EST</pubDate>
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